When you are ever in one of those moods or states where you are focusing on your height or size, you will feel bad and get negative emotions. You can look at yourself naked in them mirror and wonder why is it that you have such a short torso, or short legs, or why your head is so much bigger than your body. You look at the Cosmo and Glamour magazines and feel envy towards the women on those covers and spreads. The women all seem to have the longest legs, and the most fit perfect bodies with not even an inch of cellulite showing.

Well, I can tell you without a shadow of a doubt that as for those taut, fit bodies in the pictures are almost all photoshopped. The picture gets edited after it is taken, and then reedited to enhance certain parts of the body (i.e. breasts) and to reduce other parts of the body (i.e. love handles).

As for the dimensions that are defined by the bone, like leg length, the photoshops even work on those. A legs can be slightly lengthened using a few photoshop tricks and one who looked like they were 6′ 0″ in the previous picture now look like they are 6′ 2″.

When you look in the mirror again, ask yourself this honest question, “Would I still have this negative image of my body and my size if everyone else in the world was only 4′ 6″?”

What if you were living with a tribe in the middle of Indonesia where the indigenous people were pygmies would you still think you are “short”? Of course, not. Like most objects and phenomenon in our world, everything is relative. There is almost no absolutes in this universe. When we say that we are rich, that does not mean that we have a billion dollars on our bank account. What we really said is more like “we have more money than 95% of the rest of the society we live in”. So when we say we are tall, we are saying that our height is really in the upper percentile of the society we live in.

If you really don’t like your size, you can always just move to a country where the average height of its people are lower than where you had come from. So if you are 5′ 8″ you can move to Vietnam and you would actually be more on the tall side after living there for a short while.

My whole point of this post is to say that our perception our what we think is a immutable fact like our height/size is actually not truly immutable. While It may be true we may never be able to change ourselves, we can change our surroundings to make ourselves on the big/tall percentile side of another nation or tribe in comparison if we decided to move. So, please learn to use the concept of relativity to define your size and you can actually learn to feel better about yourself. There are plenty of people in the world who are shorter than you and if we all tried, we can all find gratitude in all the inches in height we do have, not what height we do not have.

In a previous article post I did entitled “Tallest Female Teenager In The World – Who Is It?” I had mentioned that besides the original three female contenders, there was a new addition. That was Jessica Pardoe on the UK was at the age of 19 was 6′ 9″. Now, in that article I was really trying to figure out whether a girl can still be considered a teenager after she gets past the age of 18 since 2 of the 3 top contenders were 19 or older. Only the Brazilian Elisany Silva was still technically a teenager since she should be 16 at this time (late Aug. 2012).

However, it turns out that this website seems to get quite a bit of traffic from people who are searching up the name “Jessica Pardoe” on google and other search engine sites.

Well, it was recently reported that Jessica Pardoe had her height promoted once more, to a maximum height of 6′ 10″.

Here is Jessica meeting 6′ 9″ Caroline Welz, the tallest woman in Germany. From the video it look like Caroline may actually be taller than Jessica but a look at their shoes would show that Caroline is wearing thick high heeled boots which can often give more than 2″ in extra height, and Jessica is wearing Teenager Converse-like shoes which give more like 0.5-0.75″ in extra height.

From the video on Youtube below where the story was reported by The Daily Star UK, Jessica Pardoe has grown from the mid 2011 story of her being 6′ 9″ to 6′ 10″ by only october of 2011. My real question then becomes, did Jessica really grow another inch in height from July 2011 when the news stated she was 6’9″ to 6’10” by October? If that is true then that is quite an insane growth spurt to go through.

This is just a question I always ask people who have expressed that they would like to be taller. So far in my life, I have met many family members, relatives, past girlfriends, and guy friends who have at some point proclaimed that they wished to be slightly taller.

However, for a few of these people, they were very adamant and almost obsessive about their desire to be taller. A girl I used to date who used to say that she was 5′ 1″ was really 5′ 0″ when I questioned her about it some more. When I asked her what she wished for more than anything in the world, she told me that she only wanted to be taller . I asked her about her professional dreams and what she wanted to do for a career and she didn’t have any desires and goals about them. I guess for her, she was insecure about her height.

I know that for a long time I was insecure about my height too. I tried to do some exercises and bought equipment to help me gain some extra inches (or centimeters) and I think for all of my work over 2 years, I got 0.25 of extra height, and I think I pushed from 5′ 11.75″ of height in the morning after I wake up to a height of 6′ 0″ (and slightly more) in the morning after waking up. I was very surprised the first time I measured myself and the tip of the top of my head hit the 6 foot mark. I was really glad and was not sure if it might have been a measurement error. So I double checked, and triple checked, and quadruple checked, and I found out that I had indeed grown or managed to push myself to over the 6 foot mark in height, at least in the morning after I wake up.

For me, I really wanted that extra quarter of an inch so that if I was ever on a first date with a girl and she asked me how tall I was, I could say that I was 6 foot and not lie about it. For so many girls these days, they have this strong cut off point in their own head of the minimum height their mate must be. On the internet/online dating websites, there are many women who actually set up their profile so that they can’t get emails or messages from men who are less than 6 foot tall. This means that some men on those sites will purposely lie and say that they are 6 foot when they are more like 5′ 11″ or 5′ 10″. For me, I have never used the internet or an online dating site to meet the opposite sex, but if I did, I would not want to lie about something like height. The people will eventually have to meet you, and at that moment, they know how tall you really are.

However, in the end it doesn’t matter and we all (the men and women) know that. A lot of guys like to joke that when they start doing the “horizontal tango” all issues of height disappear. I agree with these guys, but I can also point out that one can not always be doing the “horizontal tango” all the time. One does have to stand up again.

On my driver’s license when I got it first when I was 21 (I know, I was scared to drive and never took the driving test) I stated my height as 5′ 11″. After I lost that license and go a renewed license when I was 25, I decided to list my height as 6′ 0″. In the end, I am still to this day working on having good posture, walking correctly, and making sure I don’t lose the height I gained when I was younger when I get older. But that is really something that is beyond my control. For me, I do still sometimes feel insecure about my height. I feel I am too short. I feel that I am unattractive. I feel like I am not good enough. I feel like I may never be loved as much as I secretly desire inside. And all of this stuff just comes out and manifest in some bizarre behavior that other people might not understand and may find very strange.

So how about you, “Do you feel insecure about your height?”

                                             “Why are you insecure about your height?”

    “How badly do you desire to be taller than you are now?”

I have written articles talking about 7 feet tall people, 8 feet tall people, the tallest natural giants, the tallest twins, the tallest couples, the tallest toddlers, the tallest teenagers, the fattest people in history, the biggest babies in history, and everything else you could possibly think of. However that is one question that I have wanted to answer for the longest time…”Who was the largest/biggest person in history?”

Now that question is hard because now you have to consider more than one variable. When it comes to figure out who is bigger with height or weight, that is easy to do since it is one variable. To find the heaviest, you put a scale underneath them and note the number.With height, you put them next to a vertical measuring tape.

With looking for big, you have to take the two factors of height and weight together and weigh them out. In history, there has one who was just a lot “bigger” than the rest of people around. That would be Mills Darden of North Carolina.

There are no picture of this giant of a man and of the pictures on the internet, they are not of Darden himself but of someone else.

He was said to have been 7′ 6″ and weighed up to 1000 lbs. Now that is a very big man. From the Tallest Man website HERE, I paste the article on his life below…

Mills Darden – 7 feet 6 inches (228.6 cm)

Me: When it comes to these types of stories of people who were from the 19th century or any time before that when the picture and photograph had not been invented yet, it is really hard for someone ilke me to completely take the story and dimensions of this guy seriously. People back then were much smaller (shorter) and for anyone who was probably over 6′ 6″ they would have looked tremendously tall and people might have thought they were much bigger than they really are. If this guy was alive today, I would have definitely have liked to get him checked on how big he really is. Does that mean that I don’t believe this guy was as tall and heavy as the people back then proclaimed? Yeah, I would guess that this guy was not the 7’6″ height he was claimed at, but probably more like 7’0″. 

However, this is all just my own personal opinion. I still wanted to pay my respects to this person and don’t want to get into any argument of whether Mills Darden deserves the title of being called the “biggest man in history”. For this post, I really just want to state the information I found and let you interpret it how you would like.

Let’s look at the condition that is the leading cause for dwarfism, Achondroplasia. I wanted to focus more on the genetics of the disorder to understand what is the mechanism that triggers the process. From PubMed Health at National Institute Of Health HERE 

Causes, incidence, and risk factors

Achondroplasia is one of a group of disorders called chondrodystrophies or osteochondrodysplasias.

Achondroplasia may be inherited as an autosomal dominant trait, which means that if a child gets the defective gene from one parent, the child will have the disorder. If one parent has achondroplasia, the infant has a 50% chance of inheriting the disorder. If both parents have the condition, the infant’s chances of being affected increase to 75%.

However, most cases appear as spontaneous mutations. This means that two parents without achondroplasia may give birth to a baby with the condition.

In early 1994, linkage studies placed the achondroplasia gene on the short arm of human chromosome 4, distal to an anonymous marker, D4S43.
This region on chromosome 4 had been scrutinized for more than 10 years by scientists searching for the Huntington’s disease gene, and among the genes already known to reside in this area was fibroblast growth factor receptor 3 (FGFR3).
Proteins in the family of fibroblast growth factor receptors have a highly conserved structure. The protein spans the cell membrane and consists of three extracellular immunoglobulin-like domains, a lipophilic transmembrane domain, and intracellular tyrosine kinase domains. FGFR3 is expressed in cartilage and brain, and the mouse homologue is known to mediate the effect of fibroblast growth factor on chondrocytes.
By virtue of its known function and chromosomal localization, therefore, the gene encoding FGFR3 was a strong candidate for the achondroplasia gene.

Two groups of investigators have recently reported analyses of the FGFR3 gene in people with achondroplasia. Both groups found FGFR3 mutations in the DNA from affected persons and found no such mutations in the DNA from unaffected persons. In families with multiple affected members the identified mutations were inherited with the disorder. Amazingly, both groups found that every mutation was at exactly the same nucleotide in the transmembrane domain of the FGFR3 gene. In all but a very few other genetic disorders studied thus far, different affected families have different mutations in the disease gene. Shiang et al. found that 15 of the 16 achondroplasia mutations they analyzed had a guanine-to-adenine (G-to-A) transition at nucleotide 1138; the 16th mutation was a guanine-to-cytosine (G-to-C) transversion at the same nucleotide. (A point mutation is called a “transition” when a purine replaces a purine or a pyrimidine replaces a pyrimidine; it is called a “transversion” when a purine replaces a pyrimidine or vice versa.) Both mutations resulted in the substitution of arginine for glycine at amino acid 380 of the protein. Similarly, Rousseau et al. found that all 23 achondroplasia mutations in their series resulted in the same substitution at the same amino acid of the transmembrane domain of the FGFR3 protein. This high proportion of identical mutations (100 percent for the amino acid change), which is unprecedented for an autosomal dominant disorder in which more than 80 percent of cases represent new mutations, may explain the consistency of the phenotype in achondroplasia.

The achondroplasia mutations occur in a cytidine phosphate guanosine (CpG) dinucleotide in the FGFR3 gene sequence. CpG dinucleotides are known to be mutational “hot spots”; the cytosine residues adjacent to a guanine have a tendency to be methylated and then deaminated, resulting in the substitution of thymine for cytosine. This event will change a guanine to an adenine on the opposite (sense) strand which was the observed event in 37 of the 39 FGFR3 mutations reported to date. The mutation rate at nucleotide 1138 of the FGFR3 gene is therefore two to three orders of magnitude higher than the mutation rates calculated for CpG-mutation hot spots in the factor IX gene, making nucleotide 1138 the most highly mutable nucleotide currently known in the human genome. The reason for the exceptionally high mutation rate at this nucleotide, as well as the phenotypic effects of other mutations in the FGFR3 gene, remains to be explored.

Both the G-to-A transition and the G-to-C transversion at FGFR3 nucleotide 1138 create new recognition sites for restriction enzymes, making it exceptionally easy to test for the presence or absence of the mutations in genomic DNA. As a result, prenatal diagnosis of heterozygous achondroplasia, homozygous achondroplasia, and the homozygous unaffected state is now possible. The availability of prenatal diagnosis raises complex ethical issues concerning reproductive options for couples with achondroplasia. Since adults with achondroplasia are always heterozygous for the abnormal gene, it is possible for two affected parents to have children who are homozygous affected, heterozygous, or homozygous unaffected (i.e., of average stature). If only one parent is affected, the children will be either heterozygous or homozygous unaffected.

Other important advances have recently been made in our understanding of skeletal dysplasias. Hypochondroplasia, a skeletal disorder similar to but distinct from achondroplasia, seems to be linked to the same region on chromosome 4 as is achondroplasia, but no FGFR3 mutations have yet been identified in DNA from people with this condition. Reardon et al. recently reported FGFR2 mutations in patients with the Crouzon syndrome, the most common form of craniosynostosis. Through the use of positional cloning techniques, the gene for diastrophic dysplasia, an autosomal recessive skeletal dysplasia, was recently found to encode a sulfate transporter. Diastrophic dysplasia, like achondroplasia, is characterized by dwarfism, and it is especially common in Finland. Headway is being made in the identification of the genes causing several other skeletal dysplasias, including pseudoachondroplasia, multiple epiphyseal dysplasia, and cartilage hair hypoplasia. Thus, we appear to be on the threshold of a revolution in our understanding of the role of specific genes in normal and pathologic skeletal growth and development.

From MedScape Reference HERE…

Background

This question I wanted to answer is really more for the pre-epiphyseal plate closure crowd than the ones who are looking for extra height in their adult years. If you are a parent, you might be concerned and wonder whether your child will be getting the right nutrition so that they can reach their full growth and height potential. This can be a particularly worrying subject if you are of short stature yourself and so is your spouse.

We all know that on average, taller people in life are treated better, are liked more, have more romantic partners, and get more career advancement and financial rewards than their shorter counterparts. A teenager who is shorter than his/her peers and classmates may be particularly worried about this situation. If one is too short (or even too tall) they might be the victim of ridicule by others. So how does one make sure in terms of diet that they are getting everything they can to push their growth and height to the maximum?

First, we have to understand that diet makes up only a small part of the equation. What is even more important than diet is sleep and genetics. I showed in a previous post entitled”How much of human height is genetic and how much is due to nutrition?” that our genetics make up 60%-80% of what will become of our height so it is a very big factor. After that comes sleep, which is when most of our longitudinal lengthening of the long bones happen.

There is a post I had originally written entitled “The Effect On Height And Growth Being A Vegetarian“. I particularly focused on the need to get protein because the meat that vegetarians purposely choose not to eat is often the main source of protein in one’s diet. Protein is critical and essential for the human to live.

So the first thing we need to do to make sure the factor of diet is not inhibiting our growth and height is to get proteins, and as much good proteins as possible.

There is sort of a concern going around the US that most of the meat we eat came from cows, pigs, and chicken which had been fed growth hormones so that they can grow fatter faster. The fatter the animals are, the more meat there is. We american love to eat and we do had an obesity problem so it is possible that the meat we buy from the supermarket is filled with growth hormones. From a previous post I did looking at the effects of growth hormones on height, the general conclusion is that one consumes too much GH, it can theoretically speed up the growing process so that the growth plates will actually fuse earlier than they are supposed to so you end up shorter than if you had not taken the GH.

So the 2nd thing is that we need to make sure is that we are not eating meat from animals that is not been injected with GH. That is hard to do because we don’t know what type of treatment the beef we are eat is from a cow that was treated well or not. The suggestion is then to go to a place like “Whole Foods” and buy the beef that is is “grass fed”.

As for Carbohydrates, a lot of the flour made from wheat, rye, and corn these days have been processed with many forms of preservatives. Those flour are then used to make our bread, donuts, cake ,etc. In terms of how processes food will affect our overall height, I am guessing that the processed food like the flour we use to make our breads and cakes have certain compounds that are not eh healthiest in the world. The does take away from the food from having real vitamins and minerals.

However, one thing that I would say is a casual observation is that families who eat more natural food like from “Whole Foods” seem to be more on the short side. Now, I might be burned for that last comment by people who want to rebuff that with their own anecdotes and heights of people they might know and who is also big. But I am just stating an observation. We all know that foods that have not been genetically altered are usually smaller in quantity. If you have ever seen a carrot or watermelon that is grown in the wild, they look nothing like what we find in our supermarket. The fruits we see in nature are often much smaller and even a different color.

Since the cost of the stuff we buy in say “Whole Foods” can be 30-50% more expensive than the processed, genetically engineered food. I would make sense that people who are militant on only eating the most natural organic food are eating less than the people who don’t. If we wanted to correlate weight to the type of food eaten, it is obvious that people who DON’T eat organic natural food is bigger, but can we say the same about height as well. Do people who eat non-organic processed food become slightly taller than the people who don’t. That is a very hard question to answer, but I would guess it is true. So the big take away is that going in the natural organic direction may be healthier but it might be the option that leads to the greater height.

As for fiber, there is not a lot of scientific research or data that correlates to the amount of fiber we take and the height we reach. What is known is that from taking sufficient fiber, one is less likely to get slow stool or get constipation. There does seem to be some relationship with having a high fiber diet at old age with a lower mortality risk. That means that eating more vegetables has a correlation with overall health and longevity. So if you get enough fiber, your digestive system overall will be cleaner.

However, from what I have seen over my lifetime, from evolutionary theory, there seems to be an inverse correlation between longevity and height. Often it is the shorter people who outlive their taller counterparts. Taller people are more likely to develop cancer, since they have more mass in their body. The Japanese who are among the shortest group of people in the Developed world also have some of the shortest average heights of its people. In contrast, the people of the dynamic alps like Bosnia, Herzegovina, Croatia, and Serbia who have experienced so much ethnic conflict in the last century are among the tallest in the world.  They have lower lifespan than the Japanese from so much conflict but they are bigger. Of course if you even thought over my last argument, you realize that my argument is not completely logical.

In terms of fat, the human body needs fat (the good kind) to survive. Fat is made of lipids and in the human body, it is the adipose tissue. I am too lazy to do the research right now and pull out a scientific article on some study, but if I was to guess, I would say that not getting enough of the right type of fat in one’s diet will lead to a decrease in height.

Conclusion: So after going on for so long, what is the conclusion on the best type of diet needed to lead to the most growth and height? The best type of diet will involve at least 5 main parts

1. Getting all the neseccary types of vitamins and minerals, especially the type that involve in bone creation and growth and cartilage creation.

2. Getting enough of the right proteins, so eat a lot of meat like grilled chicken, fish ,grass fed cows, and eggs

3. Getting as much water as possible.

4. Eat enough fiber to have a rather clean gut to be healthier but don’t eat too much fiber.

5. Eat a lot of the good fat.

6. Try to find meat that was not injected with growth hormones

7. Eating some processed food is okay