Category Archives: Science

Grow Taller By Stretching The Vertebrate Column By Decreasing Spinal Curvature

From looking at the mechanics of how the human body functions and the degrees of freedom that is allowed by the bone structure and alignment, I personally believe that the easiest part of the human body to change which will allow for increased height would definitely be the vertebrate column.

In the human vertebrate column, there are 33 vertebrate parts, with 24 of them being moveable which are located in the upper and middle part of the torso which are separated by intervertebral disks. The other 9 is in the lower back area which are actually fused together with the upper 5 parts ath form the sacrum, and the bottom 4 parts that form the coccyx, better known as the tailbone.

However, when we are talking about adding height to our current selves, we are probably talking about stretching and elongating the upper 24 vertebrate, which actually have something between them that an be stretched out easily. The 24 vertebrate are classified by people in the medical community into 3 parts, the cervical ,the thoracic, and the lumbar part. Each part has its own curvature.

The cervical curvature and the lumbar region curves in the forward direction while the thoracic part curves in the dorsal region. That type of curvature could develop into stunted height by upwards of even 3 inches. While it is absolutely natural to have some curvature in our vertebrate column, we can still decrease the curvature to add some height without creating any significant medical condition which could result in great pain.

While I am uncertain right now whether the actual vertebrate bone can be changed and increased, it is clear that to create the spinal elongation and decrease the curvature, we will have to  change and flex the disks between the bone.

Since I am not a doctor or medical personnel, I will take this excerpt from Wikipedia to explain what exactly the intervertebral disk is made of and it’s functions.

This part is taken from the Wikipedia article for “Intervertebral disc” which can be found by clicking HERE.

Intervertebral disc

From Wikipedia, the free encyclopedia
Intervertebral discs (or intervertebral fibrocartilage) lie between adjacent vertebrae in the spine. Each disc forms a cartilaginous joint to allow slight movement of the vertebrae, and acts as a ligamentto hold the vertebrae together.

Discs consist of an outer annulus fibrosus, which surrounds the inner nucleus pulposus. The annulus fibrosus consists of several layers of fibrocartilage. The strong annular fibers contain the nucleus pulposus and distribute pressure evenly across the disc. The nucleus pulposus contains loose fibers suspended in a mucoprotein gel with the consistency of jelly. The nucleus of the disc acts as a shock absorber, absorbing the impact of the body’s daily activities and keeping the two vertebrae separated. The disc can be likened to a jelly doughnut: whereby the annulus fibrosis is similar to the dough and the nucleus pulposis is the jelly. If one presses down on the front of the doughnut the jelly moves posteriorly or to the back. When one develops a prolapsed disc the jelly/nucleus pulposus is forced out of the doughnut/disc and may put pressure on the nerve located near the disc. This can give one the symptoms of sciatica.

There is one disc between each pair of vertebrae, except for the first cervical segment, the atlas. The atlas is a ring around the roughly cone-shaped extension of the axis (second cervical segment). The axis acts as a post around which the atlas can rotate, allowing the neck to swivel. There are 23 discs in the human spine: 6 in the neck (cervical region), 12 in the middle back (thoracic region), and 5 in the lower back (lumbar region). For example, the disc between the fifth and sixth cervical vertabrae is designated “C5-6”.

Me: While this post may not give any form of method, strategy, or technique on how to achieve achieve height increase, it does provide the medical and theoretical facts that would help explain why height increase is even possible. I would guess that 90% of the cases that have shown that the person going through a program/product has definitely increased their height was probably because their vertebrate spine was elongated from them decreasing the curvature in the spine and allowing for the decompression of the spine. 

RECQL4

RECQL4 Regulates p53 Function in vivo During Skeletogenesis.

“Rothmund-Thomson syndrome, RAPADILINO, and Baller-Gerold syndrome are rare genetic disorders caused by mutations in the RECQL4 gene. These patients have significant skeletal developmental abnormalities including radial ray, limb and craniofacial defects. To investigate the role of Recql4 in the developing skeletal system, we generated Recql4 conditional knockout mice targeting the skeletal lineage. Inactivation of Recql4 using the Prx1-Cre transgene led to limb abnormalities and craniosynostosis mimicking the major bone findings in human RECQL4 patients. These Prx1-Cre+ ;Recql4fl/fl mice as well as Col2a1-Cre+ ;Recql4fl/fl mice exhibited growth plate defects and an increased p53 response in affected tissues. Inactivation of Trp53 in these Recql4 mutants resulted in genetic rescue of the skeletal phenotypes, indicating an in vivo interaction between Recql4 and Trp53, and p53 activation as an underlying mechanism for the developmental bone abnormalities in RECQL4 disorders. Our findings show that RECQL4 is critical for skeletal development by modulating p53 activity in vivo.”

“Whole skeleton preparations of P0 mice showed that the forelimbs of Prx1-Cre+;Recql4fl/fl mice were severely foreshortened and deformed. Hindlimbs of these mutants were less affected but were also smaller and shorter compared to littermate controls”

“At three weeks of age, mutant mice that survived exhibited significantly smaller limbs, as well as abnormal ossification in the joint region of the hindlimbs. Prx1-Cre+;Recql4fl/fl mutants also displayed growth retardation”

“at E18.5, H&E staining of distal femur growth plates revealed that chondrocytes from Prx1-Cre+;Recql4fl/fl mice had significantly reduced cell density and increased cell size in the resting zone (RZ), proliferating zone (PZ) and hypertrophic zone (HZ) compared to littermate controls. At three weeks of age, mutant mice showed disorganized distal femur growth plates and abnormal chondrocyte morphology demonstrating increased cell size. It appears that inactivation of Recql4 in mesenchymal progenitor cells primarily affects the growth plate chondrocytes. These cartilage phenotypes correlate with the known expression of Recql4 in the developing cartilage and may contribute to the skeletal defects in the human RECQL4 disorders at least in the appendicular skeleton.”

Deletion of Recql4 resulted in increased apoptosis and decreased proliferation of growth plate cartilage cells.  Deletion of Recql4 results in increased levels of p53 which can result in DNA damage.