This study is focused mainly on bone formation but it looks at MSCs in general and stimulating stem cells would be an important step in growing taller.

Dynamic Fluid Flow Mechanical Stimulation Modulates Bone Marrow Mesenchymal Stem Cells.

“Osteoblasts are derived from mesenchymal stem cells (MSCs){Also chondrocytes are derived from MSCs}, which initiate and regulate bone formation. New strategies for osteoporosis treatments have aimed to control the fate of MSCs. While functional disuse decreases MSC growth and osteogenic potentials, mechanical signals enhance MSC quantity and bias their differentiation toward osteoblastogenesis. Through a non-invasive dynamic hydraulic stimulation (DHS), we have found that DHS can mitigate trabecular bone loss in a functional disuse model via rat hindlimb suspension (HLS). To further elucidate the downstream cellular effect of DHS and its potential mechanism underlying the bone quality enhancement, a longitudinal in vivo study was designed to evaluate the MSC populations in response to DHS over 3, 7, 14, and 21 days. Five-month old female Sprague Dawley rats were divided into three groups for each time point: age-matched control, HLS, and HLS+DHS. DHS was delivered to the right mid-tibiae with a daily “10 min on-5 min off-10 min on” loading regime for five days/week. At each sacrifice time point, bone marrow MSCs of the stimulated and control tibiae were isolated through specific cell surface markers and quantified by flow cytometry analysis. A strong time-dependent manner of bone marrow MSC induction was observed in response to DHS, which peaked on day 14. After 21 days, this effect of DHS was diminished. the MSC pool is positively influenced by the mechanical signals driven by DHS. Coinciding with our previous findings of mitigation of disuse bone loss, DHS induced changes in MSC number may bias the differentiation of the MSC population towards osteoblastogenesis, thereby promoting bone formation under disuse conditions. the mechanism of MSC induction in response to mechanical loading [is time sensitive], and for the optimal design of osteoporosis treatments. ”

Whether MSCs are driven towards osteo or chondrogenesis could be related to the microenvironment.  So how strong the existing bone is could be a key as to whether the MSCs are driven torwards chondro or osteogenesis.

“DHS was delivered through a custom-made inflatable cuff placed around the right hind limb above the tibia.{so maybe occlusion bands could help?} An oscillatory actuator-driven syringe, a force-controlled syringe and a pressure sensor, were connected to the stimulation cuff. The actuator-driven syringe was controlled by a programmable 100 MHz waveform/signal generator. The hydraulic pressure was monitored by the pressure sensor throughout the entire treatment. With a stimulation frequency of 2 Hz, the pressure stimulation magnitudes were 30 mmHg static pressure + 30 mmHg (peak-to-peak) dynamic pressure. Daily stimulation of the “10 min on-5 min off-10 min on” loading regime was applied to each stimulated animal, while under anesthesia (isoflurane inhalation), for five days/week.”<-This is less than 0.01MPa well below the 0.1 to 10MPa to induce chondrogenesis.

Hydraulic Stimulation increased the number of bone marrow by 50%.

Dynamic fluid flow induced mechanobiological modulation of in situ osteocyte calcium oscillations.

“Distribution of intramedullary pressure (ImP) induced bone fluid flow has been suggested to influence the magnitude of mechanotransductory signals within bone. As osteocytes have been suggested as major mechanosensors in bone network, it is still unclear how osteocytes embedded within a mineralized bone matrix respond to the external mechanical stimuli derived from direct coupling of dynamic fluid flow stimulation (DFFS). While in vitro osteocytes show unique Ca(2+) oscillations to fluid shear, the objective of this study was to use a confocal imaging technique to visualize and quantify Ca(2+) responses in osteocytes in situ under DFFS into the marrow cavity of an intact ex vivo mouse femur. This study provided significant technical development for evaluating mechanotransduction mechanism in bone cell response by separation of mechanical strain and fluid flow factors using ImP stimulation, giving the ability for true real-time imaging and monitoring of bone cell activities during the stimulation. Loading frequency dependent Ca(2+) oscillations in osteocytes indicated the optimized loading at 10Hz, where such induced response was significantly diminished via blockage of the Wnt/β-catenin signaling pathway. The results provided a pilot finding of the potential crosstalk or interaction between Wnt/β-catenin signaling and Ca(2+) influx signaling of in situ osteocytes in response to mechanical signals. Findings from the present study make a valuable tool to investigate how in situ osteocytes respond and transduce mechanical signals, e.g. DFFS, as a central mechanosensor.”<-Our main concern isn’t Ca2+ oscillations as those are not likely to induce chondrogenesis directly although it’s still possible that it may have an impact or that Ca2+ could be complementary.

“Changes in the pressure or velocity of bone fluid flow (BFF) act as a communication medium that connects external loading signals and internal cellular activities in bone, which ultimately regulate the bone remodeling process”<-can we manipulate this loading signals and cellular activities in such a way that we grow taller?

“DHS[Dynamic Hydraulic Stimulation] generated local ImP that acted independently from simultaneous bone strain.”

Here’s a diagram of how Calcium signaling may alter a cell.  So Ca2+ is anabolic but it doesn’t really seem to have a direct impact on what lineage a cell traverses(osteo- versus chondro-).

Interrelation between external oscillatory muscle coupling amplitude and in vivo intramedullary pressure related bone adaptation.

“Interstitial bone fluid flow (IBFF) is suggested as a communication medium that bridges external physical signals and internal cellular activities in the bone, which thus regulates bone remodeling. Intramedullary pressure (ImP) is one main regulatory factor of IBFF and bone adaptation related mechanotransduction. Our group has recently observed that dynamic hydraulic stimulation (DHS), as an external oscillatory muscle coupling, was able to induce local ImP with minimal bone strain as well as to mitigate disuse bone loss. The current study aimed to evaluate the dose dependent relationship between DHS’s amplitude, i.e., 15 and 30mmHg, and in vivo ImP induction, as well as this correlation on bone’s phenotypic change. Simultaneous measurements of ImP and DHS cuff pressures were obtained from rats under DHS with various magnitudes and a constant frequency of 2Hz. ImP inductions and cuff pressures upon DHS loading showed a positively proportional response over the amplitude sweep. The relationship between ImP and DHS cuff pressure was evaluated and shown to be proportional, in which ImP was raised with increases of DHS cuff pressure amplitudes. A 4-week in vivo experiment using a rat hindlimb suspension model demonstrated that the mitigation effect of DHS on disuse trabecular bone was highly dose dependent and related to DHS’s amplitude, where a higher ImP led to a higher bone volume. This study suggested that sufficient physiological DHS is needed to generate ImP. Oscillatory DHS, potentially induces local fluid flow, has shown dose dependence in attenuation of disuse osteopenia. ”

“Direct ImP measurements in rats under DHS over a frequency spectrum indicated that its optimal loading at 2Hz can generate maximum ImP of 14.48±3.10mmHg. On the other hand, dynamic components with large loading amplitudes typically result in pronounced osteogenic responses”

“a micro-cardiovascular pressure transducer was carefully inserted into the tibial marrow cavity through a 1mm drilled hole, and was then tightly sealed within the drilled hole”

” the observed ImP inductions were found to be positively proportional to DHS cuff pressure, namely, which indicates promising potential of DHS loading dose dependency on ImP related bone adaptation.”

“DHS loading at 2Hz between 28.41±10.50 mmHg and 33.75±10.69 mmHg cuff pressures can induce significant ImP increases. Coincided with these observations, our 4-week in vivo study showed that DHS loading at 2Hz with 15 mmHg and 30 mmHg dynamic pressure was able to mitigate disuse trabecular bone loss in a rat functional disuse model.”

“At baseline, normal heart beats generated approximately 1mmHg tibial ImP within the marrow cavity.”

Dynamic hydraulic fluid stimulation regulated intramedullary pressure.

“Physical signals within the bone, i.e. generated from mechanical loading, have the potential to initiate skeletal adaptation. Strong evidence has pointed to bone fluid flow (BFF) as a media between an external load and the bone cells, in which altered velocity and pressure can ultimately initiate the mechanotransduction and the remodeling process within the bone. Load-induced BFF can be altered by factors such as intramedullary pressure (ImP) and/or bone matrix strain, mediating bone adaptation. Previous studies have shown that BFF induced by ImP alone, with minimum bone strain, can initiate bone remodeling. However, identifying induced ImP dynamics and bone strain factor in vivo using a non-invasive method still remains challenging. To apply ImP as a means for alteration of BFF, it was hypothesized that non-invasive dynamic hydraulic stimulation (DHS) can induce local ImP with minimal bone strain to potentially elicit osteogenic adaptive responses via bone-muscle coupling. The goal of this study was to evaluate the immediate effects on local and distant ImP and strain in response to a range of loading frequencies using DHS. Simultaneous femoral and tibial ImP and bone strain values were measured in three 15-month-old female Sprague Dawley rats during DHS loading on the tibia with frequencies of 1Hz to 10Hz. DHS showed noticeable effects on ImP induction in the stimulated tibia in a nonlinear fashion in response to DHS over the range of loading frequencies, where they peaked at 2Hz. DHS at various loading frequencies generated minimal bone strain in the tibiae. Maximal bone strain measured at all loading frequencies was less than 8με. No detectable induction of ImP or bone strain was observed in the femur. This study suggested that oscillatory DHS may regulate the local fluid dynamics with minimal mechanical strain in the bone, which serves critically in bone adaptation. These results clearly implied DHS’s potential as an effective, non-invasive intervention for osteopenia and osteoporosis treatments.”

” bone fluid flow (BFF) with altered velocity or pressure acts as a communication media between an external load and the bone cells, which then regulate bone remodeling. In converse, discontinuous BFF can initiate bone turnover and result in osteopenia”<-Initiating bone turnover could be a good thing if bone structure inhibits anabolism so lowering the frequency may be key.

“DHS was achieved through a costume-made inflatable cuff placed around the right tibia”

” the inflation and deflation of the cuff was driven by a syringe pump with the loading magnitudes and frequencies delivered by a programmable waveform/signal generator”

” 1mmHg tibial ImP and 5mmHg femoral ImP were generated by normal heart beat within the marrow cavities.”

” muscle contraction compresses the blood vessels in muscle, which generates an arteriovenous pressure gradient that further increases the hydraulic pressure in skeletal nutrient vessels and amplifies the capillary filtration in bone”

” using oscillatory muscle stimulation (MS), in which oscillatory MS induced maximal ImP at 20Hz. Oscillatory MS was achieved via two disposable needle-sized electrodes inserted into the quadriceps of the stimulated rats. The electrodes were then connected to the waveform generator with 2V supplies to induce muscle contraction”

“Due to the different physical orientations of how oscillatory MS and DHS contact the loaded tissue, as well as the different material densities and viscosities within hard and soft tissues, maximal DHS-induced ImP may result at relatively lower frequency compared to MS.  Direct hydraulic coupling may influence bone adaptation in a more physiological frequency range, where normal heart rate is 360 times per minute.”

“Changes of MSC number in response to DHS bias their differentiation towards osteoblastogenesis, leading to bone formation even under disuse conditions”

“increased bone fluid pressure and bone fluid flow regulation are strongly correlated, our current results of DHS-driven ImP induction suggest a possible mechanism that the induced ImP may subsequently enhance bone fluid flow.”

For the longest time I had a lot of respect and admiration for the great basketball player Michael Jordan. I watched him in the 90s play and reign supreme. For many guys who grew up in the 90s, MJ represented a hero that you looked up to and aspired to be like. I had thoughts of being a professional basketball player but I just wasn’t good enough. I wasn’t fast, I wasn’t tall, I wasn’t strong. Of course that is my short personal story on basketball.

What makes me think that MJ, who is often considered to be the greatest basketball player in history, has a height complex?

A few key, stories that are mentioned over and over again.

1. If you ever listened to MJ’s Hall of Fame acceptance speech, you can hear him still talk to his high school basketball coach about cutting him in the varsity team the sophomore year. MJ still had huge grievances against people who supposedly failed him when he was younger from even as back as 30 years ago. What was the real reason Jordan didn’t make it to the Varsity team his Sophomore year? – His height. At the time, MJ was around 5′ 10- 5′ 11. That position in the High School Varsity basketball team if I remember correctly went to MJ’s friend who was 6′ 8″ (named Leroy??)
2. When MJ first got into the league, he wanted Adidas to sponsor him. He had worn Adidas his whole basketball career before making it to the NBA. He wanted to be in the Adidas crowd but they actually rejected him. Instead, a little company from Oregon named Nike somehow got him to join their team, and the rest is history. Only years later did the truth come out on why Adidas skipped over him. He was not tall enough. At the time, Adidas wanted a “big man” aka center to represent them. They wanted someone like Hakeem Olajuwon. Jordan did not get the company he wanted because he was too short.
3. MJ was a extremely skilled shooting guard in UNC. He was drafted in the 1984 NBA Draft behind Olajuwon and Sam Bowie. Based on his skill alone, he should have gone at least number 2 (I personally give Hakeem respect on this point). The only reason Portland chose to go with Sam Bowie over MJ was over height again. Bowie being 7′ 1″ was a the main thing that made Portland to choose him. His size was his main attribute and what made Portland think that he was the type of player that could instantly change a franchise and make them title contenders. Portland at the time already had Clyde Drexler. They couldn’t see any point in getting another 6′ 6″ shooting guard on their team. Portland wanted a center, someone to guard the paint in the post. Portland choose height over skill that time. They would do it again in 2009 with Greg Oden with the #1 overall draft pick over the just 2 inch shorter Kevin Durant, and be bitten in the ass again for believing that height was the most important thing.
4. There is a famous documentary on MJ in the early 90s called “Come Fly With Me”. In that documentary you can hear Jordan talk about the fact that he always wanted to be 7 feet tall when he was little. He would talk about how he would hang onto the pullup bars in the playground in the backyard each day to try to grow taller. His parents saw him doing that and thought he was crazy.
5. When he was younger, he would be playing basketball against his older brother Larry, who would always beat him in the games. Larry would later only grow to around 5′ 9″. His late father James Jordan would talk about how Larry would beat Michael so often in basketball that Michael would go crazy over that.
6. MJ’s biological family is not that tall genetically. He has a sister who is only 5′ 5″ and his late father James Jordan has previously been the tallest in the family at just 6′ 0″. It was asked in the documentary how it was possible that Michael grew to be so much taller when the rest of the family was just average height. The joke the family came up with was that the milkman was tall. However, genetics came back to reveal itself when MJ’s sons from his 1st marriage to Juanita turned out to be shorter and closer to the average. One of his sons was just 6′ 2″ and the other was supposed to be 6′ 3″, as listed on their basketball player profiles at the time.
7. There is a rumor or story told from a former Bulls assistant or someone associated with the association that back in the 1995 year, Chicago was playing against Charlotte in a best of 5 game series. It was said that in game 5, when Muggsy was getting to shoot a free throw with Charlotte down just 1 point, MJ said to Muggsy “Shoot it you F**king midget”. That one sentence was the catalyst that supposedly caused the entire career of Bogue to be destroyed. The problem with this story turns out that there was no Game 5 that year. However, a recent youtube video did reveal that there was a very similar incident in game 4, where Bogue was at the free throw line and missed a very bad shot, and it does appear that MJ said something to him before that shot. Is that Muggsy Bogues story true? It is extremely possible. Let me explain. Back in the pre-season time of 2014, there was an exhibition game between the Los Angeles Lakers and Golden State Warriors in Shanghai China at the Mercedes Benz Arena. Yao Ming showed up. Muggsy Bogue did as well. Kobe did not play that game but Pau Gasol was there. Muggsy Bogue did was well. There was a famous picture taken later after the game where Yao is pictured with Muggsy and that picture is of that night.
8. A year later around Oct of 2015 at the same Arena there was another game between Charlotte and the Los Angeles Clippers.  There is a famous picture of the current MJ getting his picture taken with Patrick Ewing and Yao. MJ is standing in the center (google those pictures). In the videos that happened at the same time as that picture, MJ started to stand on his tippy-toes to correct for the height differences between him and the former centers. He might have done that as a joke but it does reveal that MJ has a huge height insecurity when he is around taller people.
9. MJ is a notorious trash talker, and it has been revealed by many people who have played against him that if the opposing guy guarding him is shorter, he would start trash-talking them about them being short, and calling them names associated with having short stature. This supposedly happened to John Starks of the Knicks. Horace Grant who is 6′ 10″, who is taller than MJ revealed in a recent podcast that MJ would be very hard on his teammates in practice. Grant admitted that MJ didn’t harass the taller guys as much, and if MJ did try to get physical with Grant like he did with Steve Kerr, Grant would have probably have gone very physical on Michael. This reveals that MJ has a very long history of harassing shorter basketball players.

It is my opinion that the basketball great Michael Jordan who lead his team to 6 NBA champions back in the 90s has always had a height complex which started from a very young age which he never has fully gotten over.

It is my personal believe and theory that one of the main reasons why he was always so competitive was because at some level MJ suffered from the “Napoleon Complex”. It might not make sense to us normal sized people since MJ is 6′ 6″ but you have to realize that when you are a professional basketball player, your height is a very critical part of your identity. If you look at the starting players on the Bulls team during this reigning era, Michael was often playing the position of Point Guard since the Shooting Guard position was often played by the equally tall 6′ 6″ Ron Harper.

Michael never got over what it felt like, the frustration that you feel, when you are playing against a person who is just taller than you , and thus can just overwhelm you based on his size alone. You sort of see this exact thing in the 1995 playoffs when the Bulls played against the Orlando Magic, who had Shaq at the time. If you look closely that the play on why the Bulls lost to the Magic in the playoffs back in 1995, you see that they just couldn’t get over the size and dominance of Shaq. There was no way that.

It doesn’t seem to matter to MJ that he was objectively tall himself because his job was to be surrounded by guys who are often much taller than him. Based on the fact that the average height of NBA players is supposed to be 6′ 7″, it would be reasonable to say that MJ would in fact be considered “short” or at least below average in height for a NBA player.

Of course, MJ would over time be able to overcome his lack of height with his disproportionately large hands.

It has been maybe 6 months since I have talked about Teplyashin and his research in his plastic surgery clinics in Russia. Before, I had said that those rumors from one weird source of some obscure online websites back in 2012 were based on truth.

That was reported by me and this very website Natural Height Growth back in 2014. We were the ones that broke that story. Since then, that news has gotten their clinic a lot of attention, and they at the clinic have noticed too, if their website directly links to our website somewhere.

Let’s look back at what has happened so far since we last talked about him. It turns out that our very own website has now been linked to the Plastic Surgery clinic in Russia somehow. At least 1 person yesterday arrived onto the website through the url beauty-plaza.ru/ru_about_professor/ based on my website analytical tracking software. People are finding our website.

I looked on this url and tried to figure out where was the link to our website or our old post. I couldn’t find it (maybe it is in the source code of the webpage somewhere or someone typed our website in through the interactive chat popup function)

Here is what I noticed very quickly. That websites has been redone, with now much more information. Also, the other websites under the clinic have also been redone. Now, there is at least 4 main websites all linked together and run in some way under Teplyashin. A year ago when I was looking up this guy, I found him mentioned a lot on the Russian media websites which I had to find through the search engine yandex.com and use the Google Translator to figure out what was he saying and what others were saying about him.

This guy is a doctor, but his original Ph. D and research is on microbiology and unique plastic surgery techniques on the human breast. (That is interesting and relevant for me because I actually spent a summer of my undergraduate career at a very prestigious University doing microbiology research for a professor whose specialty was plastic surgery.) His main source of income seems to be for giving women who come to him better looking breasts. So he runs a normal, but elite type of cosmetic surgery practice

The first question I have to ask myself it this. Did his research team really implant “neo-epiphyseal hyaline cartilage” into bone non-unions of sheep legs to see if they would grow longer? At this point, I can confidently say that he and his team did. 

However if you look at what his actual website says now, it says that the research they did on creating hyaline cartilage and mesenchymal stem cells is for the use of stem cell injections into the ends of long bones where the articular cartilage has been grounded down or deteriorated. Teplyashin’s research team has decided now to take their research and apply it for osteoarthritis applications, not height increase. It is still on cartilage regeneration and tissue engineering but the application is now shifted.

Why? It is 1000X much easier. This research that they have now written on their website and claims would be used for just makes sense. Osteoarthritis is a true serious problem that does need to be fixed. It is a condition that older people around the world suffer from which causes a lot of pain. We may be the ones complaining about being short and wishing to be taller, but there are actual people in the world who are much older than us who actually suffer through excrutiating pain.

There has been at least one person who has emailed them and contacted them asking whether their research can be applied to making people taller again. That url is now broken. Maybe it was still open back in 2013 when this website was much younger.

If you actually read the news from the stemcellrussia.com website it would tell you that it was only on April 6, 2016 that the clinic revealed that their research from the last half decade was now going to be used as a injection form to go into your leg to treat the cartilage degradation

What I can confirm to the hopefuls of using their research for height increase is that the researchers can confirmed to inquirers that their research can be used to lengthen bone. One inquirer named Talytha Sortino posted their question back in March 20, 2014. This person seemed to frequent the Make Me Taller Forum a lot and probably found my post saying that Teplyashin’s team succeeded in getting those sheep legs to be lengthened.

The person who replied back to them was Ekaterina Igorevna Kulneva, PhD, senior research at the Center of Cellular Technologies. (I used the WayBackMachine website and Chrome for automatic Google Translate)

This is what Google translated as her message

“Good Afternoon! Thank you for your interest in our company. In our center of cellular technologies developed a method for restoring bone. It is based on the implantation of biological equivalent bone created on the basis of their own stem cells. This method is effective for the recovery of large defects and bone, including for limb lengthening. The method has been successful preclinical trials.”

Another person asked a similar question. A Sebastian back in Dec 26, of 2013 asked a similar question of “I read that you guys have a stem cell method to increase…have a method to grow taller?”
I would like to cite here the fact that on the stemcellrussia.com website the website creators cite the fact that Alexander Teplyashin and his team of researchers actually wrote for industry accepted texts and medical reference books.

I refer to the Medical Reference Book “Bone Regeneration, Growth Factors, Augmentation Procedures, and Tissue Engineering Applications” – Edited by Vincent Legard and Remi Schluter. If you buy the book off of Amazon for the $240, and you turned to chapter 4. it is titled “Bone Tissue Engineering: Synthetic Materials and Stem Cells”. That specific chapter was written up by AS Teplyashin, El Kulneva, and SV Korzhikova.

The 2nd referenced book is “Stem cells and regenerative medicine” – Author: Ed. VA Tkachuk. You can refer to chapter 15 titled “Using Cellular technology in the creation of biological equivalents of bone tissue” This chapter was written by Teplyashin, Kulneva, and Korjkoff. So his team of Ph.Ds are not a group of stem cell crackpots and misfits who don’t have the resources and knowledge to get it to work out. They succeeded.

This plastic surgery and beauty clinic is completely legit. Not only that, it is not just a normal private practice plastic surgery clinic. This clinic is still doing real research, at least up to the preclinical level. What you would find most often in the USA with those Real House of Beverly Hills plastic surgeons is that those hot-shot type doctors have their own private practice making half a mil a year but they don’t do any type or real research. This is true for most doctors, who have a MD. MD means you work in the hospital and don’t really ever go back to research again, which is the job of the Ph.Ds. It is extremely rare that you find a person who has both a MD and a Ph.D perspective on things.

What I said about this guy when I found out about him back in 2014 being linked to dead fetuses found in barrels in the Ural Mountains a decade ago may be true, but that story probably had more to do with the legality of using embryo harvested stem cells after George W. Bush signed those documents back in the early 2000s banning stem cell research because he was too STEM ignorant to realize that stem cells don’t only come from embryos and unborn babies. Because of Bush’s actions from not knowing the science of stem cells well enough, our former president pushed back the progress that American scientists and researchers could have made on stem cells by at least 1 full decade, until Obama came along and reversed many of those older Bush decisions on banning stem cell research.

Alexander Teplyashin seems to be a superstar plastic surgeon that is basically known by the rich and powerful crowd of Moscow. It is possible that only the ultra rich, the billionaires and their wives/girlfriends/mistresses go see him at his clinic located somewhere in Moscow. He has been on talk shows, sat in on judge panels similar to Dancing With the Stars and America’s Got Talent as well as given hour long interviews over the radio to give his expert surgeon advice to women who want to see him to correct something about themselves cosmetically. The clinic has sent its sales reps to international legit medical conferences to present their research and findings and have filed at least 4 patents that I know of.

His clinic and his practice has been featured in dozens of magazines in Russia, usually talking about his amazing work in giving females amazing looking breasts. That is the greatest percentage of his clients. On the website you can see dozens of pictures of before and after photos of women chest, where before the breast were sagging, now the breast is perky and looks young and sexy again.

He is so famous and good of a plastic surgeon that back in Oct 21, of 2010 he was given the 1st place and voted the best foreign plastic surgeon in San Francisco.

What We Really Want and What Happened With the Stem Cell Technique?

In another source, which I will post very soon, it was revealed that his research on lengthening ram/sheeps legs was published in the newspaper “Moskovsky Komsomolets” №25512 from November 30, 2010 aka MK.RU

At the end of that article, he said to the press that they got the preclinical trails to be successful and was going to ask the Russian government permission to push forward with their goals of now going and doing clinical trials.

Let’s first define what the the term “clinical trials” even means. Clinical trials basically means in the medical industry to mean that actual humans will be experimented on. Preclinical trials are done on animals like mice, rats, pigs, monkeys, and in this case, sheep/ram.

It turns out that around the time they filed their forms and applications to start the clinical trials to test their new technique of lengthening bone on actual humans, the Russian politicians came in and reigned in the amount of stem cell research that was acceptable to be done in Russia. This was reported on the blog/news section on the website for Dec 20th, back in 2010. This is what basically stopped the research and the clinical trials from pushing forward.
This guy is actually extremely open and easy to talk to. He even offers free online consultations where you can ask him all sort os questions through Skype. Here is teplyashin’s Skype information.

username: beauty-plaza
the phone number for the clinic: +7 (495) 621 18 88

It was posted just this 16th of April (2016) in the blog/news section that every Wednesday from 1PM to 4PM Professor Teplyashin holds consultations over Skype.

The clinic have even done free consultations and competitions where females can win for a chance to get a free great lift from the clinic.
Side Note: There are some very negative websites that link to our website (sluthate, lookism, puahate) who have linked to our website since the beginning when we first started and something they have always complained about besides the height issue is that their face, is too ugly. In their heads the reason why they are having so much difficulty in finding romantic partners is only because they are too ugly and their face is something that that can’t be changed since your face is due to your facial bone structure, which has not been something you can change ever.

It turns out that in the “News” section of the Mainplastic.ru website, for Sept 28th, back in 2012 the clinic released this notice that you can now actually have bone tissue progenitor type stem cells injected into your cheeks to basically get high cheek bones.

That blog post says specifically “The slurry own bone cells injected endoscopically into the zygomatic subperiosteal region through minimal access in the temporal area. Is integrated into the bone structure, the cells begin their work – they gradually “build on” cheekbones, forming the desired and, most importantly, the forecast proportions of the face. Emphasize the natural, the perfect proportions of the face corresponding to the canons of the golden section of Leonardo da Vinci against puffy artificial persons migrating gel and lumps of fibrous tissue. The choice of our customers is obvious.”

This basically means that if one is truly insecure about one’s face, which is 90% determined by the bone structure and cartilage shape and form (This is exactly what the modeling and fashion industry has defined as beautiful. The modeling industry realizes that how attractive you are is 90% determined by the structure, form, and shape of your bone structure of your face). What this russian clinic has just said is that you can make your face more attractive with very targeted injections of bone cells to accentuate or reduce your bone structure in key areas.

Want high cheek bones? Check.
Want strong mandibular jaws like Brad Pitt? Check.
Want a strong, square chin? Check.

All of these things which you couldn’t do before is now possible, where your very own bones can be reformed and “grown” volumetrically, similar to how you form the thickness (and shape and form) of a candle by continuously dipping the original string into the wax, so that layer upon layer can slowly stack up onto each other until you get a 1 inch thick diameter of product. This is how the plastic surgeon like Teplyashin can grow your irregular bones outward, to give it definition and contours in just the right places to make your face “beautiful” .

This is probably how the Beauty Clinic run by Teplyashin can now even change your very skull bone structure and layout. Does it hurt? Minimally with a pinch like your regular botox shot maybe. A dozen sessions with the clinic and you find yourself with high cheekbones, strong jaw line, and a square chin. No surgery required. Is it that simple? I haven’t tried it myself but it could just be that simple.

Not only does his clinic say they can theoretically make you taller after your growth plates are fully closed, and make you change your facial bone structure until you become beautiful, they can even make you reverse in aging and grow younger in appearance, through the use of stem cells.

Final Note: This post will be the first of 3 posts I will be making about Teplyashin’s Research over the next few weeks because the amount of information and depth of his work is quite extensive, and from a first reading of the research they actually did, it looks like they did succeed.

If it wasn’t for the Russian politicians and their refusal to push forth with biomedical innovation, we probably might already would have had a working clinic now in Moscow offering his stem cell height increasing services for people around the world.

Could it be here already? I think so. When I guessed it would take a minimum of 15 years in the future for all the technical details to be worked out, I might be wrong. Maybe the future is already here.